Anyone running BOV with recirculation kit
10-03-2005, 09:22 AM
#2
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A number of people have run it, and its probably what >>>
I will run next. I think a lot of the Jap valves are a LOT better than the Forge - BUT I will say that I have just had a really weird time with DVs etc - I owned 2 forge valves an had issues with both. When I was on stock turbo, stock DV worked the best, and held boost fine. When I went to new turbo, straight BOV works great, except it does run rich on decel/shift.
10-03-2005, 09:43 AM
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i have a ssQ w/ recirk kit..... works great....
iv done a lot of logging w/ my vag and never noticed the car runn any richer w/ it in open bov mode or DV mode.... and i also never had the valve act up on me.... ever...
10-03-2005, 11:19 AM
#4
Only about 3 years now...>
A Diverter valve and a BOV are essentially the same thing... Classically a DV recirculates and a BOV blows off to atmoshpere, but operationally they are identical. Greddy is best known as a "BOV" but works exceptionally well in re-circ mode...
<img src="http://images5.fotki.com/v71/photos/7/7305/212841/IMG_6884a-1-vi.jpg">
Greddy S with re-circ kit:
<img src="http://images1.fotki.com/v12/photos/7/7305/158277/DSCF0008-vi.jpg">
<img src="http://images5.fotki.com/v71/photos/7/7305/212841/IMG_6884a-1-vi.jpg">
Greddy S with re-circ kit:
<img src="http://images1.fotki.com/v12/photos/7/7305/158277/DSCF0008-vi.jpg">
10-03-2005, 11:52 AM
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It's not a bad day for a monday
The Quagmire got himself suspended/deleted or whatever and boastedTT claims to be on a self-imposed break from this board.
HE got fed up dealing with US. Go figure.
HE got fed up dealing with US. Go figure.
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10-03-2005, 12:54 PM
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Could be that we've witnessed a rare brain delamination
As the Journal of Neurotrauma posits:
The loss of microvascular basal lamina antigen is known to be a consequence of cerebral ischemia, but little information is available on its role in traumatic brain injury (TBI). (J Neurotrauma. 2003 Aug;20(8):745-54.)
The loss of microvascular basal lamina antigen is known to be a consequence of cerebral ischemia, but little information is available on its role in traumatic brain injury (TBI). (J Neurotrauma. 2003 Aug;20(8):745-54.)
